About dairy tumor stomantic poisoning and fatty liver – Dairy Forum – China Animal Husbandry Website – A useful animal husbandry website
During subaracapstroposic acidosis (SARA), the rumen pH decreases every day due to the accumulation of volatile fatty acids. The survey showed the incidence of SARA between 19% and 26% in the early and medium-term lactation dairy cows. The causes of Sara include feeding excess non-structural carbohydrates and highly fermented feeds, as well as insufficient dietary crude fibers. The consequences of SARA include decline in feeding, reduced fiber digestion, germs depression, diarrhea, hoof inflammation, liver abscess, bacterial endotoxin, and inflammation characterized by increased acute protein. The increase in endotoxin in SARA-induced methods is similar, but depends on the diet that fed before induction. Even if these methods result in a similar rumen pH, the increase in acute phase proteins in SARA induction methods is also different. This indicates that the inflammatory response may not only be due to bacterial endotoxin in the rumen.
The fatty liver appears in the cow during the increase in blood non-esterified fatty acids (Nefas). The increase in blood NEFAS is related to hormonal changes in childbirth and negative energy balance. Methods to prevent fatty liver include inhibiting fatty acid mobilization in adipose tissue and altering liver metabolism to enhance fatty acid oxidation or as component output as extreme low density lipoprotein (VLDL). Nutrition and management strategies for implementing these methods have been checked. Adding the energy density of the diet by increasing the non-fibrous carbohydrate or fat, fatty liver is not prevented.
Two nutritional supplements, ruminant choline and propylene glycol have been proven to effectively prevent fatty liver. Choline may enhance the secretion of liver VLDL. Propylene glycol is most likely to reduce adipose tissue from fat. Shortening or eliminating the drying period is a management strategy that reduces the magnitude of the negative energy balance accumulated in the liver after calving.
The productive disease of the cow is caused by inconsistent with an inconsistent amount of nutrient intake, an inappropriate eating habit, an unsuitable environment, an unsuitable breeding policy or a variety of combinations of these factors. Although the transition period 3 weeks before calving is related to the peak of the morbidity of the disease, the effects of these diseases on the health and productivity of cows can extend to the following lactation period.
The latest developments of productive diseases include propylene glycol and rumen protective choline, as a supplement that prevents fatty liver, and in this case, there is no effect in which any preventive effect is increased in the close-up diet. Density prevention effect; dietIon differential (DCAD) has a linear negative impact on the incidence of emulsion, regardless of urine pH or the target level of diet DCAD; inflammatory response related to subacute rumen acidosis and its impact on feed, improve the potential of antioxidant Understanding the immunity and health status of transition period; formulating a more standardized uterine infection diagnostic criteria and treatment plan.
Existing important knowledge systems should be able to achieve the best management and prevention of cattle production diseases. One of the important challenges faced by the dairy industry is to prevent the use of the best practices of dairy productive diseases and other diseases, implementation, and economic assessments.
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