Repair course | Pig infectious atrophic rhinitis and its prevention and control plan – Yangshuo – China Animal Husbandry Website – A useful animal husbandry website

Author: Shi Zengbin
pig professional managers, micro letter ID: YZZYJLR2016
Introduction
Swine infectious atrophic rhinitis by Bordetella bronchi or toxigenic Pasteurella multocida cause A chronic respiratory infectious disease in pigs, characterized by rhinitis, face-to-face deformed, backyramine shrinkage and slow growth, clinical symptoms are sneezing, nosebleeds, face deformation, nasal skew and slow growth, increase Debao Rate, Feed The conversion rate is lowered.
I. Atrophic rhinitis (AR) classification:
Pig infectious atrophic rhinitis: all diseases that cause pig nose atrophy.
Non-carried-proof atrophic rhinitis (NPAR): BORDETELLA BRANCHISEPTICA.
Performing atrophic rhinitis (PAR): Atrin Bacillus D-type or infection with Baconobacillus. Non-carvasive diphropical (NPAR)
caused by bronchial segmentation (BB); lighter, shrinking nasal bone renewable recovery; piglets below 6 weeks old, you can see clear sneeze and “tears “; After the age of large pigs infected BB, the symptoms were slight, and the effects of growth were smaller.
A or D-type doped Bacillus (p.mult) of the PAR bistroopolysis. Light disease, or very serious (if the same is common to environmental factors and B.B); the lesion continues to exist, causing only serious clinical symptoms and growth inhibition of various daily pigs. Nasal bone atrophy and nasal bleeding.
Second, pathogen
Pathogen (1):
Bronchievania (B.B), Gram-negative rod or spherical, strictly demand oxygen. It is a pathogen of a variety of mammals. Causes the non-conductive atrophic rhinitis (NPAR) of pigs.
Bacillosin produced by bronchogenesis:
Started by Fargliobacteriosis gene (BVG) (Karen, 2000).
Skin necrotic toxin (DNT): causes nasal bone atrophy Basic factors, tracheal cytotoxins, adenosoxin toxins, osteotoxins.
Bronchiectasis: Infection Characteristics
Pathogen: Pigs with bacteria.
Dissemination: foam and feces, vertical and horizontal infection.
Easy Age: Various age, newbornPigs in early infection; for immune piggy pigs, the initial infection is related to the seriousness of the onset, the top of the pigs in the 1 week old, and the 4-week old pig is lighter, and the 9-week old pig does not have a disease. The maternal antibody can delay the infection of a piglet and a protective effect on nasal softness.
Bronchial Loss Bacillus: The pathogenic mechanism
Adhesive on nasal mucosa epithelial cell fiber, proliferation and toxin, causing the lesion of the mucosa, usually does not infect deep tissue toxin invading the nasal bone and causes bone damage The degree of pathogenism is related to the resistance of pigs and the first infection, and the nasal bone development caused by simple infection can be regenerated (under certain conditions, undermine the protective barrier of the lower respiratory tract, throwing pneumonia, and increasing the invasion of the epistemal
Pathogen (2): Pathoiscue (p.mult)
Gram-negative, rod or spherical, needle oxygen. The A or D-type P.mult of the rats induced. Strains are caused by pigs in sexual atrophic rhinitis, and the severity of lesions is proportional to toxins.
Toxosphaal toxin from the hyperlobacteria:
Skin necrotcological (DNT): DNT can cause nasal bone atrophy; directly affect P.Mult to nasal bone; important immunity Original (- “Super antigen”).
Bacillus hyperincaibacteria: infection characteristics
Pathogen: strap pig.
Dissemination: direct contact, vertical and horizontal infection.
Easy Age: Various age, newborn piglets will infect early; the age of infection can affect the severity of the onset, but the large age pig (March-age) infection also onset. A nasal dish distorted pig group is often isolated to a vikes strain. Motherborne antibodies have a protective effect on nasal soft bone disease.
Astringent Macrobilica: Avoiding Mechanism
This bacteria is generally not settled in nasal cavity, and when the environment in the nasal cavity changes (such as bronchirated Bacillus infection or chemical stimulation, etc.) Settle and proliferate and produce toxins (DNT).
Toxins degrade epithelial cells, osteoblasts were destroyed, bone dissolution, fibrous tissue replacement.
Pathogen (3): Infection mode
Bronchial loss of pedestrian bacilli is one of the common in bacteria of pigment. Through the comprehensive evaluation of different pigs (including some SPF pigs) in the world, the average of about 10% of pigs carry bronchial selared Bacillus Dunn, 1984; YOUNG Ki Choi et al, 2003). After the sow, a 1 week old fresh-raised pig nasilus can be planted in a fungus. Weaning / growth pig mix group leads to the horizontal propagation of bronchial selection.
Synergistic relationship between bronchial selection and PRRSV. – “Door Disease”
Bronchial sesametteastea and PRRSV may continue to exist in the pigs and cause subclinical diseases. Since the two have destruction of the upper respiratory barrier, the pig group is more easily infected with some conditional pathogenic bacteria (such as the use of toxin multi-killing Paste bacillus P.MULT). D / T-type toxicin Bacillus generally cause contagious atrophic rhinitis as a synergistic or secondary etiology; at the same time, in some pigs, there are places in some pigs.

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Due to the symptoms of sick rhinitis, the eye of the sick pig is inflamed, from the corner of the eye, because the tears are contained in the dust, often on the skin under the eyes, there is a half-month tear , The moist area is brown or black, so it is known as “black eyes”. AR severity affects the factors

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V. Diagnostic Method

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Anatomical variation: After slaughtering suspicious pigs, the wire between the first molars and the second molar between its heads is sawed into cross-section, observe the shape and nasal septum of the nasal bone. Change, when the nasal bone is shrinking, the curl is slightly broken, and even if there is a variety of shrinkions, even disappear, forming a hole, and the nose is bent.

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Division of lesions: Mild: ≤ 4 points;

moderate: 5–8 points;

Heavy: 9–12 points;

Pole:> 12 points.

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Sixth, AR preventive measures: vaccine + drug + environmental control 1, persistence self-cultivation;

2, eliminate sick pigs, update pigs; 3, Isolated Feeding, all sick pigs or suspected sick pigs have been separated from 3-6 months; 4, improve feeding management conditions, reduce feeding density, improve ventilation conditions, keep pig houses clean, dry, cold and warm, prevent Various stress, strictly implement the health epidemic prevention system.

5, vaccine injection, 3-4 weeks before the pigs injection of 2ml of the pregnant lagoon neck, and preventing contagious atrophic rhinitis in piglets.

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3, this is the only vaccine that can reduce the clinical signs and lesions of sexual and non-performing atrophic rhinitis.

a. Non-carried out atrophic rhinitis (NPAR): caused by the deterioration of the biopsytic bacillus. The main symptoms observed when infected with non-sexual atrophic rhinitis include: sneezing, adhesive nasal secretion, eye division, low heat, breathing, pneumonia, growth rate (ADG and FCR) and nasal bone Atrophy (reversible). These symptoms are mainly seen from the piglets around 6-7 weeks, and they can disappear after a few weeks, but the atrial bleeding of the respiratory bacteria may last for months, which may gain other lesions. Therefore, the bronchial loss of pedestrian bacilli is not only the pathogenic bacteria of NPAR, but also the susceptibility factors of other pathogen properties (and causing symptoms), such as hyperilitating Bacillus (thus causing atrophic rhinitis). Other pathogenic bacteria (such as Piginal Puertogene, Pig Bacillus, Pig Bacteria), etc. (BROCKMEIER, S. L et al. 2002; BROCKMEIER, S. L 2002; BROCKMEIER, S. L 2003; Casas, J et al. 2016), flu (Loving, Cl 2010), PRRSV (BrockMeier, S. L 2008).

b. Performing atrophic rhinitis (PAR): Toxic D-type hyperincardia is the main pathogenic bacteria, co-coercion with other infections, the most characteristic is the bronchial loss Bacillus.

The main symptoms observed when infection of sexual atrophic rhinitis include: short-jaw, nose deformed, significant growth rate (ADG and FCR) and the nasal bone severe shrink, and bend (irreversible).

4. Ruili enabled the pig group to the column, and the beneficial effects of vaccination can be observed.

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ESPHM in 2015 A study reported at the meeting (ACAL L et al. 2015) showed that immunization in the first production of the rear sow can cause a strong body fluid response, resulting in the consistent increase in the concentration of the primary milk antibody in piglets. The passive immunization of the PMT PMT for the transachase multi-killing is only until 105 days, so as to provide protection for piglets in the most critical period of the disease. Pre-premium 3-4 weeks pregnant sows were immunized once, and the immune protection time for piglets was not less than 12 weeks.

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Seven, the final conclusion

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